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Dihydromyricetin Ameliorates Experimental Ulcerative Colitis by Inhibiting Neutrophil Extracellular Traps Formation via the HIF-1α/VEGFA Signaling Pathway

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In the early stage of alveolar injury, neutrophils are recruited into the injured sites, triggering an immune response by releasing pro-inflammatory cytokines and producing neutrophil elastase (NE) to exacerbate fibrosis (Figure 1)