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Notably, enzymes participating in redox equilibrium, metabolic pathways, and signal transduction can be regulated through S-glutathionylation, a pivotal process for sustaining proper cellular functions

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Under neuroinflammatory conditions, curcumin (5-25 μM) can attenuate lipopolysaccharide-induced microglial activation, achieving approximately 60% reduction in pro-inflammatory cytokine release (tumor necrosis factor-α, interleukin-1β, interleukin-6) by suppressing the Toll-like receptor 4/myeloid differentiation primary response 88/nuclear factor kappa-B signaling cascade (Gao et al., 2019