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Finally, it is noteworthy that the data obtained from FRDA cellular models highlight an abnormal distribution of actin fibers causing defective binding of the NRF2–KEAP1 complex and the failure of NRF2 nuclear translocation [130], which may be synergistically affected by two additional mechanisms: (1) the downregulation of TRX1, which may reduce the ability of TRX1 to bind to ARE domains

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Identification of chemicals that mimic transcriptional changes associated with autism, brain aging and neurodegeneration