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Therefore, microvascular endothelium damage leads to the activation of endothelial cells with the expression of adhesion molecules on the cell surface, which promote the adhesion of leukocytes and platelets, leading to further changes in perfusion and oxygen delivery, and additional amplification of ROS generation, tissue injury and inflammatory response [7,43]

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Characterization of RIPK3-mediated phosphorylation of the activation loop of MLKL during necroptosis

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