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Understanding the metabolic programming of neural progenitors and the role of mitochondrial metabolism in neurogenesis provides insights into the pathology of mitochondrial diseases and offers potential therapeutic avenues for LS, addressing significant unmet medical needs in pediatric patient

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There have been reports of abnormal protein expression of Prx isoforms in the brains of neurodegenerative diseases such as Alzheimer’s disease (AD), frontotemporal dementia, Parkinson’s disease (PD), amyotrophic lateral sclerosis (ALS), and Huntington’s disease (HD) [53,54,55,56,57]