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Furthermore, the susceptibility of WT or Cys-to-Ser mutants of GLRX to H 2 O 2 - or GSSG-mediated inactivation was evaluated by measuring the reaction of GLRX with eosin-labeled GSSG or eosin-conjugated glutathionylated BSA, to model physiological substrates

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Novel therapeutic strategies targeting fibroblasts to improve heart disease