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With the use of the ASOs to target the SCN1A NAT, one study was effectively able to rescue the sense SCN1A gene expression, ameliorate neuronal excitability and seizures in a mouse model of DS—an effect that almost entirely recapitulates the rescued phenotypes observed via CRISPRa treatment [74, 75]

Antioxidant that Protects Cells from Oxidative Damage

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From the data in Figure 3(b), the cells treated with different concentration presented different migration rates and healing progression