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[DOI] [PubMed] [Google Scholar] 152.Talebi S., Arab A., Sorraya N

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For example, transient, low-level increases in IGF-1 signaling in endothelial cells may augment eNOS activity and preserve vasodilator function early in disease, whereas sustained or cell-type-specific overactivation drives proliferative, anti-apoptotic programs later in disease progression (Jin et al., 2014

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