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In transgenic mouse models of amyloidosis, the ABCC1 transport activity is increased to promote GSH release, possibly as a protective mechanism against oxidative stress, although this mechanism is not sustained in the long-term period (Zoufal et al., 2020)

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Subsequently, the nodal explants were moved to MS supplemented with 2.5 μM NAA and 2.5 μM IBA for 20 days to induce root formation