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Research has shown that BAs in combination with its activating transcription factor, farnesoid X receptor (FXR), inhibit cholesterol transport and increase cholesterol excretion by macrophages in order to reduce cholesterol concentration in plasma, and can be used as a therapeutic target for dyslipidemia (Figure 2

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These findings support previously published results showing that also on a genetic level, ferroptosis-related protein expression in the brain differs between patients who died early compared to later stages of AD pathology [15]