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Supporting these observations, Sangaunchom and Dharmasaroja demonstrated that caffeine potentiates ethanol-induced neurotoxicity by further inhibiting the mTOR/p70S6K/4E-BP1 axis in neuronal cells, underscoring the central role of mTOR dysregulation in ethanol-related apoptotic vulnerability [66]

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These neurochemical changes lead to problems with reward processing, reduced motivational importance, impaired cognitive flexibility, and alterations in synaptic plasticity