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Cellular interactions and molecular mechanisms in the pathogenesis of NDMA-induced hepatic fibrosis A schematic representation of the cellular interactions and molecular mechanisms involved in the pathogenesis of NDMA-induced hepatic fibrosis is presented in Fig

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Additional research considerations include characterizing whether the enhanced membrane permeability of the acetylated form alters tissue distribution in in vivo models, whether the modified charge distribution affects receptor or chromatin binding dynamics, and whether the extended half-life requires adjusted concentration protocols in cell culture systems