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In the acute phase, METH causes an excessive release of dopamine and glutamate, resulting in transient deficits in attention and working memory (Mark et al., 2004)

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Interestingly, the treatment of MRC-5 fibroblasts with 20 mM of taurine has appeared to inhibit the lipopolysaccharide (LPS)-induced ROS formation and the activation of the MAPK signaling pathway (272)