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As a class of irreversible common blinding diseases (8), glaucoma is characterized by optic nerve damage and visual field loss, and its pathogenesis is complex, involving multiple factors such as genetics, environment, and lifestyle (9–11)

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Collectively, current evidence indicates that hyperuricemia-associated inflammation is driven by a complex network involving intestinal immune dysregulation, systemic cytokine release, and activation of innate immune signaling pathways such as NF-κB and the NLRP3 inflammasome