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Also, SIRT1 may stimulate the expression of Bcl-2 through the deacetylation of FOXO (77, 78)

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The peptide works through several mechanisms: it upregulates growth factor expression (particularly VEGF), promotes angiogenesis (the formation of new blood vessels at injury sites) [5,12] , modulates the nitric oxide system, and supports the FAK-paxillin pathway, which governs cell migration to damaged tissue