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On one hand, insulin promotes glycolysis, improves glucose utilization, inhibits hepatic glycogenolysis and gluconeogenesis, and suppresses hepatic glucose output (Saltiel and Kahn, 2001

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Its homologs have been found in rats and mice in different brain regions (prefrontal cortex, hippocampus, thalamic nuclei, brainstem, and mesencephalic regions), as well as in peripheral tissues such as the spleen, adrenal glands, and jejunum [55]